Test on Macular Diseases: Diagnosis and Treatment
Macular Diseases: Diagnosis and Treatment for Students
Test: Epiretinal membrane, Macular hole: Vitreous traction syndromes & adhesion, Macular hole: Classification and staging, Macular hole: Evaluation and management, Macular telangiectasia, OCT imaging, Cystoid macular edema, Central serous chorioretinopathy, Surgical technique, Case series
20 questions
Question 1: The provided case study confirms a diagnosis of Type 2 Idiopathic Macular Telangiectasia (MCT).
A. Ano
B. Ne
Explanation: The conclusion of the abstract explicitly states, "It also was confirmed that confirmation of a diagnosis of Type 2 MCT in primary polycystic diagnosed with AMD."
Question 2: Based on the provided study materials, which of the following are advantages of Swept-source OCT (SS-OCT) for choroid visualization?
A. It enables the visualization of the whole thickness of the choroid.
B. It allows for faster and deeper visibility of structures.
C. It uses a tunable laser with wavelengths of 1050-1060 nm for wider imaging.
D. It is an older technology compared to spectral domain OCT and provides limited choroid views.
Explanation: The study materials state that 'Swept source optical coherence tomography has enabled the visualization of the whole thickness of the choroid', indicating option 0 is correct. It also notes 'Faster and deeper visibility of structures' and '1050-10660 nm and faster scanning like 100-400 scans per second for a deeper, faster, and wide imaging', confirming options 1 and 2. The materials explicitly mention SS-OCT was introduced in 2012 and is 'Much better even than ED' and spectral domain is 'spectral domainolder', making option 3 incorrect.
Question 3: Postsurgical CME patients on topical NSAID and steroid therapy are typically followed up after 8 weeks to determine their response to the treatment.
A. Ano
B. Ne
Explanation: The Wills Manual states that postsurgical CME patients should be followed up in 4 to 6 weeks to determine their response to topical drop therapy.
Question 4: According to the study materials, which of the following mediators contribute to tissue inflammation, increased vasodilation, and vasopermeability, thereby inducing Cystoid Macular Edema (CME)?
A. Anti-VEGFs
B. Prostaglandins
C. Calcium 1
D. VEGF
Explanation: The study materials state, 'Many mediators disrupt the blood – aqueous barrier (and blood – retinal barrier), leading to increased vascular permeability. Any disease process that can break down these barriers can induce CME Mediators such as prostaglandins contribute to tissue inflammation, increasing vasodilation and vasopermeability. Like prostaglandins bc they are mediators of inflammation'.
Question 5: Pigment Epithelial Detachment (PED) is listed as a differential diagnosis for Central Serous Chorioretinopathy (CSCR).
A. Ano
B. Ne
Explanation: The study materials explicitly list PED as a differential diagnosis (DDx) for CSCR under the 'Differentials in CSCR' section.